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Atp6ap2 deletion causes extensive vacuolation that consumes the insulin content of pancreatic β cells
Katrina J. Binger*, Martin Neukam, Sudhir Gopal Tattikota, Fatimunnisa Qadri, Dmytro Puchkov, Diana M. Willmes, Sabrina Wurmsee, Sabrina Geisberger, Ralf Dechend, Klemens Raile, Thomas Kurth, Genevieve Nguyen, Matthew N. Poy, Michele Solimena, Dominik N. Muller, Andreas L. Birkenfeld
*Corresponding author for this work
- La Trobe University
- Bio21 Molecular Science and Biotechnology Institute
- University Hospital Carl Gustav Carus
- Harvard Medical School
- Max-Delbrück-Center for Molecular Medicine
- Institute for Radiology, CCM
- Forschungsinstitut für Molekulare Pharmakologie (FMP)
- TU Dresden
- Collège de France
- Johns Hopkins University
- Molecular Diabetology
- Department of Diabetology Endocrinology and Nephrology
- University Hospital Tübingen
- Department of Therapy of Diabetes
- University of Tuebingen
- Section of Diabetes and Nutritional Sciences
- UCL University College London
Research output: Contribution to journal › Article › peer-review
24
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(Scopus)