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Deletion of the diabetes candidate gene Slc16a13 in mice attenuates diet-induced ectopic lipid accumulation and insulin resistance
Tina Schumann, Jörg König, Christian von Loeffelholz, Daniel F. Vatner, Dongyan Zhang, Rachel J. Perry, Michel Bernier, Jason Chami, Christine Henke, Anica Kurzbach, Nermeen N. El-Agroudy, Diana M. Willmes, Dominik Pesta, Rafael de Cabo, John F. O´Sullivan, Eric Simon, Gerald I. Shulman, Bradford S. Hamilton, Andreas L. Birkenfeld*
*Corresponding author for this work
- Dresden University School of Medicine
- University Hospital Carl Gustav Carus
- German Center for Diabetes Research
- Friedrich-Alexander-University of Erlangen-Nürnberg
- University Hospital of Jena
- Yale University
- National Institute on Aging
- Heart Research Institute Australia
- German Aerospace Center (DLR), Institute of Aerospace Medicine
- University Hospital Cologne
- Leibniz Center for Diabetes Research at Heinrich Heine University Düsseldorf
- University of Sydney
- Royal Prince Alfred Hospital
- Boehringer Ingelheim GmbH
- Section of Metabolic and Vascular Medicine
- Departments of Diabetes
- King's College London
- University of Tuebingen
- Department of Endocrinology
- University Hospital Tübingen
Research output: Contribution to journal › Article › peer-review
13
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(Scopus)