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ELAC2 Mutations Cause a Mitochondrial RNA Processing Defect Associated with Hypertrophic Cardiomyopathy
Tobias B. Haack, Robert Kopajtich, Peter Freisinger, Thomas Wieland, Joanna Rorbach, Thomas J. Nicholls, Enrico Baruffini, Anett Walther, Katharina Danhauser, Franz A. Zimmermann, Ralf A. Husain, Jessica Schum, Helen Mundy, Ileana Ferrero, Tim M. Strom, Thomas Meitinger, Robert W. Taylor, Michal Minczuk*, Johannes A. Mayr, Holger Prokisch
*Corresponding author for this work
- Helmholtz Zentrum München GmbH
- Klinikum Reutlingen, Dept Pediat
- MRC, Mitochondrial Biol Unit
- University of Parma
- Paracelsus Medical University Nuremberg
- Jena Univ Hosp, Friedrich Schiller University of Jena, Dept Neuropediatr
- German Center for Cardiovascular Research
- Munich Heart Alliance
- Newcastle University
- Tech Univ Munich
Research output: Contribution to journal › Article › peer-review
112
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(Scopus)