Diapedesis-Induced Integrin Signaling via LFA-1 Facilitates Tissue Immunity by Inducing Intrinsic Complement C3 Expression in Immune Cells

Martin Kolev, Erin E. West, Natalia Kunz, Daniel Chauss, E. Ashley Moseman, Jubayer Rahman, Tilo Freiwald, Maria L. Balmer, Jonas Lötscher, Sarah Dimeloe, Elizabeth C. Rosser, Lucy R. Wedderburn, Katrin D. Mayer-Barber, Andrea Bohrer, Paul Lavender, Andrew Cope, Luopin Wang, Mariana J. Kaplan, Niki M. Moutsopoulos, Dorian McGavernSteven M. Holland, Christoph Hess, Majid Kazemian^*, Behdad Afzali, Claudia Kemper

^*Corresponding author for this work

Inflammation Biology

Complement and Inflammation Research Section (CIRS)
National Heart, Lung, and Blood Institute (NIH)
National Institute of Diabetes and Digestive and Kidney Diseases (NIH)
Neuroscience Center, National Institute of Neurological Disorders and Stroke, Bethesda, Maryland, USA.
University of Basel
Birmingham Business School
UCL University College London
NIAID National Institute of Allergy and Infectious Diseases, National Institutes of Health
School of Immunology and Microbial Sciences
King's College London
Purdue University
NIAMS - National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIH)
National Institute of Dental and Craniofacial Research (NIH)
Cambridge University Hospitals NHS Foundation Trust
Institute for Systemic Inflammation Research
Institut für Integrative und ExperimentelleGenomik

Research output: Contribution to journal › Article › peer-review

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Diapedesis-Induced Integrin Signaling via LFA-1 Facilitates Tissue Immunity by Inducing Intrinsic Complement C3 Expression in Immune Cells

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Immunology and Microbiology